The following sections of this BookRags Literature Study Guide is offprint from Gale's For Students Series: Presenting Analysis, Context, and Criticism on Commonly Studied Works: Introduction, Author Biography, Plot Summary, Characters, Themes, Style, Historical Context, Critical Overview, Criticism and Critical Essays, Media Adaptations, Topics for Further Study, Compare & Contrast, What Do I Read Next?, For Further Study, and Sources.
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The following sections, if they exist, are offprint from Beacham's Encyclopedia of Popular Fiction: "Social Concerns", "Thematic Overview", "Techniques", "Literary Precedents", "Key Questions", "Related Titles", "Adaptations", "Related Web Sites". (c)1994-2005, by Walton Beacham.
The following sections, if they exist, are offprint from Beacham's Guide to Literature for Young Adults: "About the Author", "Overview", "Setting", "Literary Qualities", "Social Sensitivity", "Topics for Discussion", "Ideas for Reports and Papers". (c)1994-2005, by Walton Beacham.
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Rigor mortis (from the Latin for "stiffness of death") is the rigidity that develops in a body after death. This rigidity may begin shortly after death--within 10 to 15 minutes--or may not begin until several hours later, depending on the condition of the body at the time of death and on environmental factors, such as moisture content of the air and particularly temperature. A colder temperature promotes a slower onset of rigor mortis.
Typically, rigor mortis affects facial muscles first. Spreading to other parts of the body follows. The body will remain fixed in the rigid position until decomposition of tissue begins, about 24 to 48 hours after death.
Rigor mortis occurs because metabolism continues in muscles for a short while after death. As part of the metabolic activity, adenosine triphosphate (ATP) is produced from the metabolism of a sugar compound called glycogen. ATP is a principal energy source for muscular activity. So, as long as it is present, muscles continue to maintain their tone. As the store of glycogen is exhausted, ATP can no longer be made and its concentration decreases. One of the consequences of ATP depletion is the formation of abnormal links between two components of muscle tissue, actin and myosin. The leakage of calcium into the muscle cells also contributes to the formation of abnormal actin-myosin links. The abnormality produces the stiffening of the muscle, which persists until the links are decomposed.